In re Compensation of Maldonado, 102519 ORWC, 14-04365

Case DateOctober 25, 2019
CourtOregon
71 Van Natta 1226 (2019)
In the Matter of the Compensation of RAMON M. MALDONADO, Claimant
WCB No. 14-04365
Oregon Worker Compensation
October 25, 2019
          Dunn & Roy PC, Claimant Attorneys           SAIF Legal Salem, Defense Attorneys           Reviewing Panel: Members Curey and Ousey. [1]          ORDER ON REMAND          This matter is before the Board on remand from the Court of Appeals. SAIF v. Maldonado, 294 Or.App. 252 (2018). The court has reversed the Board's order, Ramon M. Maldonado, 68 Van Natta, that had reversed an Administrative Law Judge's (ALJ's) order that upheld the SAIF Corporation's denial of claimant's new/omitted medical condition claim for a worsened low back spondylolytic defect/spondylolysis condition. Reasoning that the Board erred in applying the “law of the case” doctrine in analyzing the persuasiveness of physician opinions on which SAIF had relied, the court has remanded for a determination of whether such evidence is persuasive without application of the aforementioned doctrine. Having received the parties' briefs on remand, we proceed with our review.          FINDINGS OF FACT          On February 28, 2013, claimant sustained a compensable low back injury when he shook dirt off a 50-pound tree root ball and experienced the sudden onset of low back pain. (Ex. 3). X-rays showed L5 grade 1 spondylolisthesis with suspected spondylolysis and degenerative changes at L5-S1. (Ex. 4).          In April 2013, Dr. Mohabeer, an occupational medicine physician, diagnosed lumbar conditions including sprain/strain, herniated disc, spinal stenosis, spondylolisthesis, and radiculopathy. (Exs. 8, 11-5). He opined that the February 2013 work injury was the major contributing cause of claimant's current condition/need for treatment. (Id.)          An MRI showed L3-4 canal stenosis, bilateral L5 neural foraminal stenosis, bilateral L5 spondylolysis and grade 1 spondylolisthesis, posterior annular bulges/ protrusions at all lumbar levels, and posterior tears at L2-3 and L4-5. (Ex. 10). Electrodiagnostic studies reflected no neurological abnormalities. (Ex. 13).          [71 Van Natta 1227] In May 2013, Dr. Brett, orthopedic surgeon, diagnosed an L3-4 disc protrusion and worsened L5-S1 spondylolytic spondylolisthesis with bilateral L5 impingement. (Ex. 14-1). He noted preexisting L5-S1 conditions, but opined that the February 2013 injury was the major contributing factor in the development of his L3-4 cauda equina compression, bilateral L5 radiculitis, and radiculopathy at L5-S1 “resulting in pathologic worsening with his lifting incident of 02/28/2013.” (Ex. 14-3).          On follow-up, Dr. Mohabeer concluded that the MRI showed multiple posttraumatic lumbar pathologies, including L5-S1 herniated disc, spondylolisthesis, spinal stenosis, and nerve root impingement. (Exs. 11-5, 15-5). He correlated these findings to claimant's current work-related symptomatology. (Ex. 11-5).          Dr. Duff, orthopedic surgeon, performed an examination at SAIF's request. (Ex. 18). He diagnosed an acute lumbar strain related to the February 2013 injury, in addition to multilevel degenerative disc disease, L4-5 and L5-S1 central and foraminal stenosis, and L5-S1 grade 1 spondylolisthesis. (Ex. 18-5). He opined that the imaging study findings were idiopathic and degenerative, “with the spondylolisthesis being an added development anomaly frequently associated with back complaints in middle age.” (Ex. 18-6).          In June 2013, Dr. Brett determined that claimant had an annular injury, L3-4 disc herniation, and probable pathological worsening of his preexisting spondylolytic spondylolisthesis at L5-S1. (Ex. 24).          In August 2013, Dr. Vessely, orthopedic surgeon, performed an examination at SAIF's request. (Ex. 29). He diagnosed L5-S1 spondylolisthesis, significant multilevel spondylosis, and a February 2013 work injury with inconsistencies. (Ex. 29-8). He did not find any “acute changes in [claimant's] lumbar spine that [he] would relate to an injury process.” (Ex. 29-10). Dr. Vessely determined that claimant's conditions were related to degeneration, genetics, and natural aging. (Id.) He did not believe that claimant's condition and clinical status had any relationship to what occurred on February 28, 2013. (Ex. 29-10, -11). He opined that claimant's work incident did not meet the “material level of causation, ” and concluded that there was no “combined condition.” (Ex. 29-11).          In mid-August, 2013, Dr. Sweeney, chiropractor, signed a concurrence from claimant's counsel, agreeing that claimant's MRI showed degenerative, preexisting spondylosis and a pars defect, which he described as a fracture of part of the vertebrae and not acutely caused by the work injury. (Ex. 32-2, -3). [71 Van Natta 1228]          In mid-August 2013, Dr. Mohabeer signed a concurrence from claimant's counsel, diagnosing L5-S1 spondylolisthesis, which was a slippage of the vertebrae and caused pinching of a nerve. (Ex. 33-2). He indicated that the condition was not surgical or symptomatic until it likely worsened with claimant's work injury, causing further slippage. (Id.) Finally, Dr. Mohabeer stated that claimant's “worsened spondylolysis in the lower back, was caused in major part by his work injury.” (Ex. 33-3).          Dr. Brett also signed a concurrence letter from claimant's counsel, opining that claimant had “spondylosis at L5-S1 which allowed the L5 disc to slip forward onto the S1 vertebrae.” (Ex. 34-2). He indicated that “this spondylolysis was the breakage of bones and likely was a congenital or abnormality that was present at birth * * *.” (Id.) He noted that the MRI showed significant slippage, and concluded that claimant's work injury worsened his “spondylolisthesis, ” which caused it to further slide forward. (Id.) He explained that the traumatic L5-S1 disc protrusion “actually allow[ed] that slippage to occur more rapidly and it was a combination of the disc protrusion and the spondylolisthesis that caused the impingement of the bilateral L5 nerve roots at that level.” (Id.) Dr. Brett concluded that “the major contributing cause of that combination of the preexisting spondylolysis, the progression of the spondylolisthesis, the degenerative changes and the disc protrusion, [was claimant's] work activities as occurred on 02/28/2013 [.]” (Ex. 34-3).          In October 2013, pursuant to an approved stipulation, SAIF agreed to accept the claim for lumbar strain/sprain, L3-4 disc protrusion, and L5-S1 disc protrusion. (Ex. 39). SAIF issued a Notice of Acceptance accepting those conditions. (Ex. 40).          In November 2013, claimant began treating with Dr. Blake, physiatrist, who did not detect objective findings of nerve root impingement at any level on either side of the lumbar spine. (Ex. 42-4). In December 2013, Dr. Blake noted that Dr. Collada, neurosurgeon, had reviewed claimant's medical records and concluded that he did not have evidence of a surgical problem. (Ex. 43-1). Dr. Blake referred claimant to Dr. Rosenbaum, neurosurgeon, for another opinion.2 (Exs. 44-1, 45).          [71 Van...

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